Heart rhythm disorders temporally associated with obstructive apnoeic events have been highlighted for more than 30 years. An ECG from polysomnographic recordings enables both sleep medicine specialists and cardiologists to address occasional very impressive paroxysmal atrioventricular conduction blocks or sinus pauses (of up to 10 s during rapid eye movement (REM) sleep) [1]。这些呼吸事件引起的这些疾病的病理生理显然很复杂,在人类综合生理学的背景下代表了令人兴奋的挑战。急性和慢性失衡在自主神经系统(ANS)中与阻塞性睡眠呼吸暂停或脑力下(OSA)相关的作用已在心肌适应这种重复应激的许多细胞机制中清楚地证明了[OSA)[2-4]。此外,这些自主疾病现在被认为是独立的危险因素或心脏衰竭患者突然心脏死亡的触发因素[5],但也有保留的收缩/舒张/舒张左心室功能的人[6,,,,7]。尽管睡眠通常是心脏副交感神经调节的时期,而心肌电稳定性得到增强,但OSA却打扰了这种静止,从而产生了自主神经特征,在这种自主神经方面,在这种自主神经方面,在这种自主神经方面都会常见的心动性活动,导致心动过低和交感神经过度活跃,有利于心室的心脏偏心。8]。
审查论文的作者发表在当前的期刊上188bet体育备用网址欧洲呼吸杂志[[9)必须祝贺成功的详细说明of the mechanisms present during apnoea/hypopnoea that can lead to heart rhythm disturbances. This paper is, for me, a remarkable teaching tool that I will use again in the future due to its clarity and its “updated” literature review.
在第二部分…