@article {yan684，作者= {yan，s和sinderby，c和bielen，p and p and Beck，j and comtois，n和sliwinski，p}，title = {慢性阻塞性肺部疾病的呼气肌肉压力和呼吸机制，体积，体积= {16}，number = {4}，pages = {684--690}，ear = {2000}，publisher = {欧洲呼吸社188bet官网地址会}，摘要= {呼气肌肉招募在稳定的慢性阻塞性肺部疾病中很常见（COPD（COPD）） 患者。由于气道阻塞，几乎没有理由相信COPD中的主动到期将在降低操作肺部容积方面有效。因此，COPD中呼气肌肉募集的生理意义仍然未知。这项研究的目的是评估COPD患者在静止时呼吸，呼气性肌肉收缩对隔膜产生能力的影响。从其压力摆动（PDI）评估了给定的隔膜电活动（EDI）的隔膜产生能力，其中EDI被标准化为其最大值的\％（PDI/EDI/EDI，MAX）。阶段呼气肌肉收缩是胃压（PGA，Exp.Rise）的总呼气升高。中度至重度COPD的19例座位的患者参加了研究，10例患者在平均PGA到期期间表现出PGA的阶段性上升，Exp.Rise为1.91 +/- 0.89 CMH2O。因此，将患者分为被动到期（PE）和主动到期（AE）组。两组之间的各种肺功能和呼吸模式参数没有显着差异。 Pdi/Edi/Edi,max was 0.63+/-0.07 and 0.54+/-0.07 cmH2O/\% in PE and AE groups, respectively, and was not significantly different between each other. Compared with PE group, AE group not only recruited expiratory muscles, but also preferentially recruited inspiratory rib cage muscles and derecruited the diaphragm. The results do not support a significant improvement of the force-generating ability of the diaphragm by phasic contraction of expiratory muscles at rest in chronic obstructive pulmonary disease patients.}, issn = {0903-1936}, URL = {//www.qdcxjkg.com/content/16/4/684}, eprint = {//www.qdcxjkg.com/content/16/4/684.full.pdf}, journal = {European Respiratory Journal} }